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New results published in a recent issue of Nature Immunology show that the mutant NOD2 gene leads to a lowered production of IL-10. Before now, it was unclear how the mutation influences Crohn's in humans, because mice with the mutation produce normal levels of IL-10. Cells harvested from human patients with the mutation were found to produce less IL-10 in laboratory experiments. The researchers hope their findings will eventually lead to therapies that can overcome the IL-10 deficiency in Crohn's patients. Subscribe to our FREE Ezine and be eligible for Health News, discounted products/services and coupons related to your Health. We publish 24/7. HealthNewsDigest.com Top of Page 
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